Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters
Identifieur interne : 000049 ( Main/Corpus ); précédent : 000048; suivant : 000050Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters
Auteurs : Riccardo Riccardi ; Fiorenzo Caita ; Carla Ciustetto ; Silvia CardiolSource :
- Pacing and Clinical Electrophysiology [ 0147-8389 ] ; 1997-05.
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Abstract
Clinical electrophysiology has not yet clearly defined atrial features that can predict spontaneous occurrence of atrial fibrillation (AF). The aim of this work was to identify atrial electrophysiological features that can distinguish Wolff‐Parkinson‐White patients with spontaneous AF from those without this arrhythmia. Sixty‐nine patients with Wolff‐Parkinson‐White were divided into three groups: group I (16 patients) with spontaneous AF; group II (35 patients) with reciprocating tachycardia but not AF; and group III (18 patients) asymptomatic without documented arrhythmias. Atrial effective refractory periods (ERPs) and intraatrial conduction times in response to premature extrastimuli were analyzed. The latter were evaluated as the A1A2 interval minus the correspondent S1S2 interval (A1A2‐S1S2), S2A2 and the interval A1A2 following the shortest S1S2 producing atrial activation (FRP'). All the parameters have been evaluated in two atrial sites and at two atrial pacing cycle lengths (600 and 400 ms). For all the parameters, the difference (“gradient”) was calculated between the values of the same parameter measured at the atrial pacing cycle length of 600 ms and that found at the atrial pacing cycle length of 400 ms in the same recording site in each patient was calculated. Atrial FRP did not differ significantly in the three groups. Intraatrial conduction parameters, evaluated in the high right atrium (HRA), were longer when measured at an atrial pacing of 400 ms and showed a lack of rate adaptation in patients with spontaneous AF. In group I patients in particular, FRP’became longer with the increase of atrial rate, while in groups 2 and 3, it usually shortened. The mean gradient of HRA FRP’was ‐15.0 ± 19 ms in group I as compared to 5.7 ±13 ms in group II and 6.4± 13 ms in group III (P < 0.001); sensitivity. specificity, and negative predictive value of a negative gradient in the identification of patients with spontaneous AF, were, respectively, 83%, 75%, and 93%. Patients from groups 2 and 3 did not differ in any of the analyzed parameters. Patients with Wolff‐Parkinson‐White and spontaneous AF showed prolonged intraatrial conduction times and a different behavior in response to modification of heart rate. (PACE 1997;20[Pt. I]:1318‐1327)
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DOI: 10.1111/j.1540-8159.1997.tb06786.x
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<record><TEI wicri:istexFullTextTei="biblStruct"><teiHeader><fileDesc><titleStmt><title xml:lang="en">Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters</title><author><name sortKey="Riccardi, Riccardo" sort="Riccardi, Riccardo" uniqKey="Riccardi R" first="Riccardo" last="Riccardi">Riccardo Riccardi</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author><author><name sortKey="Caita, Fiorenzo" sort="Caita, Fiorenzo" uniqKey="Caita F" first="Fiorenzo" last="Caita">Fiorenzo Caita</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author><author><name sortKey="Ciustetto, Carla" sort="Ciustetto, Carla" uniqKey="Ciustetto C" first="Carla" last="Ciustetto">Carla Ciustetto</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author><author><name sortKey="Cardiol, Silvia" sort="Cardiol, Silvia" uniqKey="Cardiol S" first="Silvia" last="Cardiol">Silvia Cardiol</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author></titleStmt><publicationStmt><idno type="wicri:source">ISTEX</idno><idno type="RBID">ISTEX:94C5B038421723AD74D864A8E8EAEC4955412D6E</idno><date when="1997" year="1997">1997</date><idno type="doi">10.1111/j.1540-8159.1997.tb06786.x</idno><idno type="url">https://api.istex.fr/document/94C5B038421723AD74D864A8E8EAEC4955412D6E/fulltext/pdf</idno><idno type="wicri:Area/Main/Corpus">000049</idno></publicationStmt><sourceDesc><biblStruct><analytic><title level="a" type="main" xml:lang="en">Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters</title><author><name sortKey="Riccardi, Riccardo" sort="Riccardi, Riccardo" uniqKey="Riccardi R" first="Riccardo" last="Riccardi">Riccardo Riccardi</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author><author><name sortKey="Caita, Fiorenzo" sort="Caita, Fiorenzo" uniqKey="Caita F" first="Fiorenzo" last="Caita">Fiorenzo Caita</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author><author><name sortKey="Ciustetto, Carla" sort="Ciustetto, Carla" uniqKey="Ciustetto C" first="Carla" last="Ciustetto">Carla Ciustetto</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author><author><name sortKey="Cardiol, Silvia" sort="Cardiol, Silvia" uniqKey="Cardiol S" first="Silvia" last="Cardiol">Silvia Cardiol</name><affiliation><mods:affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</mods:affiliation></affiliation></author></analytic><monogr></monogr><series><title level="j">Pacing and Clinical Electrophysiology</title><idno type="ISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1997-05">1997-05</date><biblScope unit="volume">20</biblScope><biblScope unit="issue">5</biblScope><biblScope unit="page" from="1318">1318</biblScope><biblScope unit="page" to="1327">1327</biblScope></imprint><idno type="ISSN">0147-8389</idno></series><idno type="istex">94C5B038421723AD74D864A8E8EAEC4955412D6E</idno><idno type="DOI">10.1111/j.1540-8159.1997.tb06786.x</idno><idno type="ArticleID">PACE1318</idno></biblStruct></sourceDesc><seriesStmt><idno type="ISSN">0147-8389</idno></seriesStmt></fileDesc><profileDesc><textClass><keywords scheme="KwdEn" xml:lang="en"><term>Wolff‐Parkinson‐White</term><term>atrial fibrillation</term><term>atrial refractoriness</term><term>electrophysiological study</term><term>intraatrial conduction</term></keywords></textClass><langUsage><language ident="en">en</language></langUsage></profileDesc></teiHeader><front><div type="abstract" xml:lang="en">Clinical electrophysiology has not yet clearly defined atrial features that can predict spontaneous occurrence of atrial fibrillation (AF). The aim of this work was to identify atrial electrophysiological features that can distinguish Wolff‐Parkinson‐White patients with spontaneous AF from those without this arrhythmia. Sixty‐nine patients with Wolff‐Parkinson‐White were divided into three groups: group I (16 patients) with spontaneous AF; group II (35 patients) with reciprocating tachycardia but not AF; and group III (18 patients) asymptomatic without documented arrhythmias. Atrial effective refractory periods (ERPs) and intraatrial conduction times in response to premature extrastimuli were analyzed. The latter were evaluated as the A1A2 interval minus the correspondent S1S2 interval (A1A2‐S1S2), S2A2 and the interval A1A2 following the shortest S1S2 producing atrial activation (FRP'). All the parameters have been evaluated in two atrial sites and at two atrial pacing cycle lengths (600 and 400 ms). For all the parameters, the difference (“gradient”) was calculated between the values of the same parameter measured at the atrial pacing cycle length of 600 ms and that found at the atrial pacing cycle length of 400 ms in the same recording site in each patient was calculated. Atrial FRP did not differ significantly in the three groups. Intraatrial conduction parameters, evaluated in the high right atrium (HRA), were longer when measured at an atrial pacing of 400 ms and showed a lack of rate adaptation in patients with spontaneous AF. In group I patients in particular, FRP’became longer with the increase of atrial rate, while in groups 2 and 3, it usually shortened. The mean gradient of HRA FRP’was ‐15.0 ± 19 ms in group I as compared to 5.7 ±13 ms in group II and 6.4± 13 ms in group III (P < 0.001); sensitivity. specificity, and negative predictive value of a negative gradient in the identification of patients with spontaneous AF, were, respectively, 83%, 75%, and 93%. Patients from groups 2 and 3 did not differ in any of the analyzed parameters. Patients with Wolff‐Parkinson‐White and spontaneous AF showed prolonged intraatrial conduction times and a different behavior in response to modification of heart rate. (PACE 1997;20[Pt. I]:1318‐1327)</div></front></TEI><istex><corpusName>wiley</corpusName><author><json:item><name>RICCARDO RICCARDI</name><affiliations><json:string>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</json:string></affiliations></json:item><json:item><name>FIORENZO CAITA</name><affiliations><json:string>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</json:string></affiliations></json:item><json:item><name>CARLA CIUSTETTO</name><affiliations><json:string>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</json:string></affiliations></json:item><json:item><name>SILVIA CARDIOL</name><affiliations><json:string>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</json:string></affiliations></json:item></author><subject><json:item><lang><json:string>eng</json:string></lang><value>atrial fibrillation</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>Wolff‐Parkinson‐White</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>intraatrial conduction</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>electrophysiological study</value></json:item><json:item><lang><json:string>eng</json:string></lang><value>atrial refractoriness</value></json:item></subject><articleId><json:string>PACE1318</json:string></articleId><language><json:string>eng</json:string></language><abstract>Clinical electrophysiology has not yet clearly defined atrial features that can predict spontaneous occurrence of atrial fibrillation (AF). The aim of this work was to identify atrial electrophysiological features that can distinguish Wolff‐Parkinson‐White patients with spontaneous AF from those without this arrhythmia. Sixty‐nine patients with Wolff‐Parkinson‐White were divided into three groups: group I (16 patients) with spontaneous AF; group II (35 patients) with reciprocating tachycardia but not AF; and group III (18 patients) asymptomatic without documented arrhythmias. Atrial effective refractory periods (ERPs) and intraatrial conduction times in response to premature extrastimuli were analyzed. The latter were evaluated as the A1A2 interval minus the correspondent S1S2 interval (A1A2‐S1S2), S2A2 and the interval A1A2 following the shortest S1S2 producing atrial activation (FRP'). All the parameters have been evaluated in two atrial sites and at two atrial pacing cycle lengths (600 and 400 ms). For all the parameters, the difference (“gradient”) was calculated between the values of the same parameter measured at the atrial pacing cycle length of 600 ms and that found at the atrial pacing cycle length of 400 ms in the same recording site in each patient was calculated. Atrial FRP did not differ significantly in the three groups. Intraatrial conduction parameters, evaluated in the high right atrium (HRA), were longer when measured at an atrial pacing of 400 ms and showed a lack of rate adaptation in patients with spontaneous AF. In group I patients in particular, FRP’became longer with the increase of atrial rate, while in groups 2 and 3, it usually shortened. The mean gradient of HRA FRP’was ‐15.0 ± 19 ms in group I as compared to 5.7 ±13 ms in group II and 6.4± 13 ms in group III (P > 0.001); sensitivity. specificity, and negative predictive value of a negative gradient in the identification of patients with spontaneous AF, were, respectively, 83%, 75%, and 93%. Patients from groups 2 and 3 did not differ in any of the analyzed parameters. Patients with Wolff‐Parkinson‐White and spontaneous AF showed prolonged intraatrial conduction times and a different behavior in response to modification of heart rate. (PACE 1997;20[Pt. 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Fax: 39‐141‐392220.</p></note><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation></author><author><persName><forename type="first">FIORENZO</forename><surname>CAITA</surname></persName><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation></author><author><persName><forename type="first">CARLA</forename><surname>CIUSTETTO</surname></persName><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation></author><author><persName><forename type="first">SILVIA</forename><surname>CARDIOL</surname></persName><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation></author></analytic><monogr><title level="j">Pacing and Clinical Electrophysiology</title><idno type="pISSN">0147-8389</idno><idno type="eISSN">1540-8159</idno><idno type="DOI">10.1111/(ISSN)1540-8159</idno><imprint><publisher>Blackwell Publishing Ltd</publisher><pubPlace>Oxford, UK</pubPlace><date type="published" when="1997-05"></date><biblScope unit="volume">20</biblScope><biblScope unit="issue">5</biblScope><biblScope unit="page" from="1318">1318</biblScope><biblScope unit="page" to="1327">1327</biblScope></imprint></monogr><idno type="istex">94C5B038421723AD74D864A8E8EAEC4955412D6E</idno><idno type="DOI">10.1111/j.1540-8159.1997.tb06786.x</idno><idno type="ArticleID">PACE1318</idno></biblStruct></sourceDesc></fileDesc><profileDesc><creation><date>1997</date></creation><langUsage><language ident="en">en</language></langUsage><abstract xml:lang="en"><p>Clinical electrophysiology has not yet clearly defined atrial features that can predict spontaneous occurrence of atrial fibrillation (AF). The aim of this work was to identify atrial electrophysiological features that can distinguish Wolff‐Parkinson‐White patients with spontaneous AF from those without this arrhythmia. Sixty‐nine patients with Wolff‐Parkinson‐White were divided into three groups: group I (16 patients) with spontaneous AF; group II (35 patients) with reciprocating tachycardia but not AF; and group III (18 patients) asymptomatic without documented arrhythmias. Atrial effective refractory periods (ERPs) and intraatrial conduction times in response to premature extrastimuli were analyzed. The latter were evaluated as the A1A2 interval minus the correspondent S1S2 interval (A1A2‐S1S2), S2A2 and the interval A1A2 following the shortest S1S2 producing atrial activation (FRP'). All the parameters have been evaluated in two atrial sites and at two atrial pacing cycle lengths (600 and 400 ms). For all the parameters, the difference (“gradient”) was calculated between the values of the same parameter measured at the atrial pacing cycle length of 600 ms and that found at the atrial pacing cycle length of 400 ms in the same recording site in each patient was calculated. Atrial FRP did not differ significantly in the three groups. Intraatrial conduction parameters, evaluated in the high right atrium (HRA), were longer when measured at an atrial pacing of 400 ms and showed a lack of rate adaptation in patients with spontaneous AF. In group I patients in particular, FRP’became longer with the increase of atrial rate, while in groups 2 and 3, it usually shortened. The mean gradient of HRA FRP’was ‐15.0 ± 19 ms in group I as compared to 5.7 ±13 ms in group II and 6.4± 13 ms in group III (P < 0.001); sensitivity. specificity, and negative predictive value of a negative gradient in the identification of patients with spontaneous AF, were, respectively, 83%, 75%, and 93%. Patients from groups 2 and 3 did not differ in any of the analyzed parameters. Patients with Wolff‐Parkinson‐White and spontaneous AF showed prolonged intraatrial conduction times and a different behavior in response to modification of heart rate. (PACE 1997;20[Pt. I]:1318‐1327)</p></abstract><textClass xml:lang="en"><keywords scheme="keyword"><list><head>Keywords</head><item><term>atrial fibrillation</term></item><item><term>Wolff‐Parkinson‐White</term></item><item><term>intraatrial conduction</term></item><item><term>electrophysiological study</term></item><item><term>atrial refractoriness</term></item></list></keywords></textClass></profileDesc><revisionDesc><change when="1997-05">Published</change></revisionDesc></teiHeader></istex:fulltextTEI><json:item><original>false</original><mimetype>text/plain</mimetype><extension>txt</extension><uri>https://api.istex.fr/document/94C5B038421723AD74D864A8E8EAEC4955412D6E/fulltext/txt</uri></json:item></fulltext><metadata><istex:metadataXml wicri:clean="Wiley, elements deleted: body"><istex:xmlDeclaration>version="1.0" encoding="UTF-8" standalone="yes"</istex:xmlDeclaration><istex:document><component version="2.0" type="serialArticle" xml:lang="en"><header><publicationMeta level="product"><publisherInfo><publisherName>Blackwell Publishing Ltd</publisherName><publisherLoc>Oxford, UK</publisherLoc></publisherInfo><doi origin="wiley" registered="yes">10.1111/(ISSN)1540-8159</doi><issn type="print">0147-8389</issn><issn type="electronic">1540-8159</issn><idGroup><id type="product" value="PACE"></id><id type="publisherDivision" value="ST"></id></idGroup><titleGroup><title type="main" sort="PACING CLINICAL ELECTROPHYSIOLOGY">Pacing and Clinical Electrophysiology</title></titleGroup></publicationMeta><publicationMeta level="part" position="05005"><doi origin="wiley">10.1111/pace.1997.20.issue-5</doi><numberingGroup><numbering type="journalVolume" number="20">20</numbering><numbering type="journalIssue" number="5">5</numbering></numberingGroup><coverDate startDate="1997-05">May 1997</coverDate></publicationMeta><publicationMeta level="unit" type="article" position="0131800" status="forIssue"><doi origin="wiley">10.1111/j.1540-8159.1997.tb06786.x</doi><idGroup><id type="unit" value="PACE1318"></id></idGroup><countGroup><count type="pageTotal" number="10"></count></countGroup><eventGroup><event type="firstOnline" date="2006-06-30"></event><event type="publishedOnlineFinalForm" date="2006-06-30"></event><event type="xmlConverted" agent="Converter:BPG_TO_WML3G version:2.3.2 mode:FullText source:HeaderRef result:HeaderRef" date="2010-03-02"></event><event type="xmlConverted" agent="Converter:WILEY_ML3G_TO_WILEY_ML3GV2 version:3.8.8" date="2014-02-06"></event><event type="xmlConverted" agent="Converter:WML3G_To_WML3G version:4.1.7 mode:FullText,remove_FC" date="2014-11-03"></event></eventGroup><numberingGroup><numbering type="pageFirst" number="1318">1318</numbering><numbering type="pageLast" number="1327">1327</numbering></numberingGroup><correspondenceTo>Address for reprints: Riccardo Riccardi, M.D., Via Servais 200 A/18 10146 Torino, Italy. Fax: 39‐141‐392220.</correspondenceTo><linkGroup><link type="toTypesetVersion" href="file:PACE.PACE1318.pdf"></link></linkGroup></publicationMeta><contentMeta><unparsedEditorialHistory>Received April 7, 1995; revision September 27, 1995; accepted February 2, 1996.</unparsedEditorialHistory><countGroup><count type="referenceTotal" number="33"></count><count type="linksCrossRef" number="5"></count></countGroup><titleGroup><title type="main">Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters</title></titleGroup><creators><creator creatorRole="author" xml:id="cr1" affiliationRef="#a1" corresponding="yes"><personName><givenNames>RICCARDO</givenNames><familyName>RICCARDI</familyName></personName></creator><creator creatorRole="author" xml:id="cr2" affiliationRef="#a1"><personName><givenNames>FIORENZO</givenNames><familyName>CAITA</familyName></personName></creator><creator creatorRole="author" xml:id="cr3" affiliationRef="#a1"><personName><givenNames>CARLA</givenNames><familyName>CIUSTETTO</familyName></personName></creator><creator creatorRole="author" xml:id="cr4" affiliationRef="#a1"><personName><givenNames>SILVIA</givenNames><familyName>CARDIOL</familyName></personName></creator></creators><affiliationGroup><affiliation xml:id="a1" countryCode="IT"><unparsedAffiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</unparsedAffiliation></affiliation></affiliationGroup><keywordGroup xml:lang="en"><keyword xml:id="k1">atrial fibrillation</keyword><keyword xml:id="k2">Wolff‐Parkinson‐White</keyword><keyword xml:id="k3">intraatrial conduction</keyword><keyword xml:id="k4">electrophysiological study</keyword><keyword xml:id="k5">atrial refractoriness</keyword></keywordGroup><abstractGroup><abstract type="main" xml:lang="en"><p>Clinical electrophysiology has not yet clearly defined atrial features that can predict spontaneous occurrence of atrial fibrillation (AF). The aim of this work was to identify atrial electrophysiological features that can distinguish Wolff‐Parkinson‐White patients with spontaneous AF from those without this arrhythmia. Sixty‐nine patients with Wolff‐Parkinson‐White were divided into three groups: group I (16 patients) with spontaneous AF; group II (35 patients) with reciprocating tachycardia but not AF; and group III (18 patients) asymptomatic without documented arrhythmias. Atrial effective refractory periods (ERPs) and intraatrial conduction times in response to premature extrastimuli were analyzed. The latter were evaluated as the A<sub>1</sub>A<sub>2</sub> interval minus the correspondent S<sub>1</sub>S<sub>2</sub> interval (A<sub>1</sub>A<sub>2</sub>‐S<sub>1</sub>S<sub>2</sub>), S<sub>2</sub>A<sub>2</sub> and the interval A<sub>1</sub>A<sub>2</sub> following the shortest S<sub>1</sub>S<sub>2</sub> producing atrial activation (FRP'). All the parameters have been evaluated in two atrial sites and at two atrial pacing cycle lengths (600 and 400 ms). For all the parameters, the difference (“gradient”) was calculated between the values of the same parameter measured at the atrial pacing cycle length of 600 ms and that found at the atrial pacing cycle length of 400 ms in the same recording site in each patient was calculated. Atrial FRP did not differ significantly in the three groups. Intraatrial conduction parameters, evaluated in the high right atrium (HRA), were longer when measured at an atrial pacing of 400 ms and showed a lack of rate adaptation in patients with spontaneous AF. In group I patients in particular, FRP’became longer with the increase of atrial rate, while in groups 2 and 3, it usually shortened. The mean gradient of HRA FRP’was ‐15.0 ± 19 ms in group I as compared to 5.7 ±13 ms in group II and 6.4± 13 ms in group III (P < 0.001); sensitivity. specificity, and negative predictive value of a negative gradient in the identification of patients with spontaneous AF, were, respectively, 83%, 75%, and 93%. Patients from groups 2 and 3 did not differ in any of the analyzed parameters. Patients with Wolff‐Parkinson‐White and spontaneous AF showed prolonged intraatrial conduction times and a different behavior in response to modification of heart rate. (PACE 1997;20[Pt. I]:1318‐1327)</p></abstract></abstractGroup></contentMeta></header></component></istex:document></istex:metadataXml><mods version="3.6"><titleInfo lang="en"><title>Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters</title></titleInfo><titleInfo type="alternative" contentType="CDATA" lang="en"><title>Atrial Electrophysiological Features in Patients with Wolff‐Parkinson‐White and Atrial Fibrillation: Absence of Rate Adaptation of Intraatrial Conduction Time Parameters</title></titleInfo><name type="personal"><namePart type="given">RICCARDO</namePart><namePart type="family">RICCARDI</namePart><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation><description>Correspondence: Address for reprints: Riccardo Riccardi, M.D., Via Servais 200 A/18 10146 Torino, Italy. Fax: 39‐141‐392220.</description><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">FIORENZO</namePart><namePart type="family">CAITA</namePart><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">CARLA</namePart><namePart type="family">CIUSTETTO</namePart><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><name type="personal"><namePart type="given">SILVIA</namePart><namePart type="family">CARDIOL</namePart><affiliation>From the Division of Cardiology, Ospedale Civile di Asti, and the Division of Cardiology, University of Torino, Torino, Italy</affiliation><role><roleTerm type="text">author</roleTerm></role></name><typeOfResource>text</typeOfResource><genre type="article" displayLabel="article"></genre><originInfo><publisher>Blackwell Publishing Ltd</publisher><place><placeTerm type="text">Oxford, UK</placeTerm></place><dateIssued encoding="w3cdtf">1997-05</dateIssued><edition>Received April 7, 1995; revision September 27, 1995; accepted February 2, 1996.</edition><copyrightDate encoding="w3cdtf">1997</copyrightDate></originInfo><language><languageTerm type="code" authority="rfc3066">en</languageTerm><languageTerm type="code" authority="iso639-2b">eng</languageTerm></language><physicalDescription><internetMediaType>text/html</internetMediaType><extent unit="references">33</extent></physicalDescription><abstract lang="en">Clinical electrophysiology has not yet clearly defined atrial features that can predict spontaneous occurrence of atrial fibrillation (AF). The aim of this work was to identify atrial electrophysiological features that can distinguish Wolff‐Parkinson‐White patients with spontaneous AF from those without this arrhythmia. Sixty‐nine patients with Wolff‐Parkinson‐White were divided into three groups: group I (16 patients) with spontaneous AF; group II (35 patients) with reciprocating tachycardia but not AF; and group III (18 patients) asymptomatic without documented arrhythmias. Atrial effective refractory periods (ERPs) and intraatrial conduction times in response to premature extrastimuli were analyzed. The latter were evaluated as the A1A2 interval minus the correspondent S1S2 interval (A1A2‐S1S2), S2A2 and the interval A1A2 following the shortest S1S2 producing atrial activation (FRP'). All the parameters have been evaluated in two atrial sites and at two atrial pacing cycle lengths (600 and 400 ms). For all the parameters, the difference (“gradient”) was calculated between the values of the same parameter measured at the atrial pacing cycle length of 600 ms and that found at the atrial pacing cycle length of 400 ms in the same recording site in each patient was calculated. Atrial FRP did not differ significantly in the three groups. Intraatrial conduction parameters, evaluated in the high right atrium (HRA), were longer when measured at an atrial pacing of 400 ms and showed a lack of rate adaptation in patients with spontaneous AF. In group I patients in particular, FRP’became longer with the increase of atrial rate, while in groups 2 and 3, it usually shortened. The mean gradient of HRA FRP’was ‐15.0 ± 19 ms in group I as compared to 5.7 ±13 ms in group II and 6.4± 13 ms in group III (P < 0.001); sensitivity. specificity, and negative predictive value of a negative gradient in the identification of patients with spontaneous AF, were, respectively, 83%, 75%, and 93%. Patients from groups 2 and 3 did not differ in any of the analyzed parameters. Patients with Wolff‐Parkinson‐White and spontaneous AF showed prolonged intraatrial conduction times and a different behavior in response to modification of heart rate. (PACE 1997;20[Pt. I]:1318‐1327)</abstract><subject lang="en"><genre>Keywords</genre><topic>atrial fibrillation</topic><topic>Wolff‐Parkinson‐White</topic><topic>intraatrial conduction</topic><topic>electrophysiological study</topic><topic>atrial refractoriness</topic></subject><relatedItem type="host"><titleInfo><title>Pacing and Clinical Electrophysiology</title></titleInfo><genre type="Journal">journal</genre><identifier type="ISSN">0147-8389</identifier><identifier type="eISSN">1540-8159</identifier><identifier type="DOI">10.1111/(ISSN)1540-8159</identifier><identifier type="PublisherID">PACE</identifier><part><date>1997</date><detail type="volume"><caption>vol.</caption><number>20</number></detail><detail type="issue"><caption>no.</caption><number>5</number></detail><extent unit="pages"><start>1318</start><end>1327</end><total>10</total></extent></part></relatedItem><identifier type="istex">94C5B038421723AD74D864A8E8EAEC4955412D6E</identifier><identifier type="DOI">10.1111/j.1540-8159.1997.tb06786.x</identifier><identifier type="ArticleID">PACE1318</identifier><recordInfo><recordContentSource>WILEY</recordContentSource><recordOrigin>Blackwell Publishing Ltd</recordOrigin></recordInfo></mods></metadata><serie></serie></istex></record>Pour manipuler ce document sous Unix (Dilib)
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